Environmental Epigenetics by L. Joseph Su, Tung-chin Chiang

By L. Joseph Su, Tung-chin Chiang

This ebook examines the toxicological and well-being implications of environmental epigenetics and offers wisdom via an interdisciplinary process. integrated during this quantity are chapters outlining numerous environmental hazard components equivalent to phthalates and nutritional elements, lifestyles states reminiscent of being pregnant and getting old, hormonal and metabolic concerns and particular illness hazards reminiscent of melanoma cardiovascular illnesses and different non-communicable diseases.

Environmental Epigenetics imparts integrative wisdom of the technological know-how of epigenetics and the problems raised in environmental epidemiology.

This publication is meant to serve either as a reference compendium on environmental epigenetics for scientists in academia, and laboratories and as a textbook for graduate point environmental health and wellbeing courses.

Environmental Epigenetics imparts integrative wisdom of the technological know-how of epigenetics and the problems raised in environmental epidemiology.

This booklet is meant to serve either as a reference compendium on environmental epigenetics for scientists in academia, and laboratories and as a textbook for graduate point environmental well-being courses.

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Nature 445(7125):257–258 Chapter 3 Epigenetic Changes in Hormonal Related Disease: Uterine Leiomyoma (Fibroids) Tung-Chin Chiang Abstract Epigenetic modifications have been associated with etiology of hormonal related diseases such as breast cancer and prostate cancer. In this chapter, the epigenetic status of a less studied hormonal related disease, uterine leiomyoma, was investigated and its application in potential therapeutic was further discussed. Differential methylation status, the most studied epigenetic signatures, is associated with differential gene expression.

For example, in the late 1960s, cross-sectional studies were used during the outbreak of blackfoot disease and reported associations of arsenic in drinking water and blackfoot disease (Tseng 1977; Tseng et al. 1968). It is relatively quick, inexpensively, and less time-consuming to conduct cross-sectional study. The association of multiple exposures and disease outcomes can be examined at the same time and therefore can be used as hypothesis generation. More rigorous study design such as cohort study or randomized control trial should be followed to test the hypothesis.

Radiother Oncol 111(2):168–177 Steves CJ, Spector TD, Jackson SH (2012) Ageing, genes, environment and epigenetics: what twin studies tell us now, and in the future. Age Ageing 41(5):581–586 Stratton MR, Campbell PJ, Futreal PA (2009) The cancer genome. Nature 458(7239):719–724 Su LJ et al (2011) Epigenetic contributions to the relationship between cancer and dietary intake of nutrients, bioactive food components, and environmental toxicants. Front Genet 2:91 Sun M et al (2008) Curcumin (diferuloylmethane) alters the expression profiles of microRNAs in human pancreatic cancer cells.

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