Biochemical Aspects of Nervous Diseases by J. Wilson, R. H. S. Thompson (auth.), J. N. Cumings (eds.)

By J. Wilson, R. H. S. Thompson (auth.), J. N. Cumings (eds.)

Within contemporary years simple wisdom in regards to the chemistry, and the metabolic techniques occurring within the mind, spinal twine, peripheral nerves and muscular tissues has elevated in a impressive demeanour. therefore a few extremely important books were released either in the United States and in Europe during which a few, a minimum of, of this data has turn into to be had in an simply readable shape to an ever expanding team of laboratory and clinical staff. the applying of such reports in quite a few neurological illnesses can now be made, therefore making attainable a proof of the various scientific and pathological peculiarities which have been recognized for many years. even if a number of small manuals have already been released, combining either biochemical and medical points of such problems and those were studied by means of neurologists and chemical pathologists through the international, but a quantity dedicated completely to the biochemistry of neurological illnesses has no longer but been on hand. the current quantity is an try to treatment this omission in terms of quite a few of the stipulations. person writers of every of the six chapters were selected who're in detail involved either with biochemistry and with its program to affliction in guy. each one writer has been liable for the accuracy of his bankruptcy including acceptable references from the literature, however the Editor doesn't inevitably concur with the entire critiques expressed by means of the authors.

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E. step-wise degradation from the opposite end of the molecule, might be blocked in Refsum's disease. Their early results did indeed suggest that there was such a defect; they showed that in patients with Refsum's disease there was a reduction in the excretion of sebacic acid (aC lO-dicarboxylic acid) after a loading dose of tricaprin [121]. However, these preliminary results were of uncertain significance because their patients did not show defects in w-oxidation of other substrates [130]. Later they showed that in patients whose serum phytanic acid was reduced by dietary treatment, the tricaprin test became normal [131], and in fact there is no direct evidence that w-oxidation is of importance in phytanic acid metabolism.

Intemat. Copenhagen, p. 885 (1939) 24. J. Fennelly, O. Frank, H. Baker and C. M. Leevy, Brit. med. , 2, 1290 (1964) 25. R. W. Gilliatt in Biochemical Aspects of Neurological Disorders U. N. Cumings and M. Kremer, eds), p. 117, Blackwell Scientific Publications, Oxford (1965) 26. P. K. Thomas and R. G. Lascelles, Lancet, i, 1355 (1965) 27. P. K. Thomas and R. G. Lascelles, Quart. j. , 35,489 (1966) 28. R. Goodhart and H. M. Sinclair,j. bioi. , 132, 11 (1940) 29. M. M. Martin, Brain, 76,594 (1953) 30.

E. step-wise degradation from the opposite end of the molecule, might be blocked in Refsum's disease. Their early results did indeed suggest that there was such a defect; they showed that in patients with Refsum's disease there was a reduction in the excretion of sebacic acid (aC lO-dicarboxylic acid) after a loading dose of tricaprin [121]. However, these preliminary results were of uncertain significance because their patients did not show defects in w-oxidation of other substrates [130]. Later they showed that in patients whose serum phytanic acid was reduced by dietary treatment, the tricaprin test became normal [131], and in fact there is no direct evidence that w-oxidation is of importance in phytanic acid metabolism.

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